Although
intraocular pressure remains as the most important risk factor for the
development of glaucoma, and lowering it is the only clinically validated
method of treating the disease, there is enough evidence to support the
existence of other factors that may contribute to glaucomatous damage. The
mechanisms involved in the pathogenesis of glaucoma are not fully elucidated
and probably include pressure-dependent and pressure-independent pathways. This
presentation will discuss the rationale and the potential clinical application
of blood flow improvement, neuroprotection, and genetic therapy as additional
tools in the treatment of glaucoma.