TOXIC EFFECTS OF MITOMYCIN-C ON TRABECULAR MESHWORK AND CILIARY PROCESS CELLS

*Kwou-Yeung Wu, Show-Jen Hong and *Hwei-Zu Wang

Department of *Ophthalmology and Pharmacology, Kaohsiung Medical University, Kaohsiung, Taiwan

INTRODUCTION: Prolonged postoperative hypotony has been a common and serious complication of glaucomatous filtration surgery combined with mitomycin-C. In this study, the toxic effects of mitomycin-C in cultured ciliary process and trabecular meshwork cells in vitro were investigated. METHODS: The toxic effects of mitomycin-C to cultured rabbit ciliary process and trabecular meshwork cells were estimated by MTT method and 3H-thymidine uptake after 3 min, 5 min and 60 min treatment. The changes of intracellular Ca+2 in trabecular meshwork cells and cAMP in ciliary process cells were measured. DNA fragmentation was also performed on both tissues which were excised from rabbit eyes. RESULTS: Mitomycin-C is more toxic to ciliary process cells than to trabecular meshwork cells estimated by MTT and 3H-thymidine uptake. Mitomycin-C at 10-3 mg/ml could inhibit 50 mM KCl-induced calcium influx into trabecular meshwork cells. The cAMP concentration in ciliary process cells was also inhibited by 10-3 mg/ml mitomycin-C after 3 min and 5 min exposure. In the presence of 10-3 mg/ml mitomycin-C for 3 min and 5 min, DNA fragmentation was significantly observed in ciliary process tissue but not in trabecular meshwork tissue. CONCLUSION: These findings indicate that post-filtration hypotony may result from the toxicity of mitomycin-C to ciliary epithelial cells and trabecular meshwork cells.